JURANYI ZSOLT PDF

Jurányi Zsolt is the author of Az alvilág zsoldjában ( avg rating, 16 ratings, 1 review, published ) and Az alvilág csapdájában ( avg rating. nov. Az alvilág csapdájában has 3 ratings and 1 review. Sándor said: Az első rész fényévekkel jobb volt, szerintem. Csak azért vergődtem végig. List of computer science publications by Zsolt Jurányi.

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Neurochemical ResearchVol.

Alterations in brain extracellular dopamine and jurwnyi levels following combined administration of the glycine transporter type-1 inhibitor Org and risperidone. Combined drug administration with D2 uuranyi receptor blockade and activation of hypofunctional NMDA receptors may be needed for a more effective treatment of positive and negative symptoms and the accompanied cognitive deficit in schizophrenia.

AB – The most dominant hypotheses for the pathogenesis of schizophrenia have focused primarily upon hyperfunctional dopaminergic and hypofunctional glutamatergic neurotransmission in the central nervous system.

Link to citation list in Scopus. Interestingly, the extracellular concentrations of glutamate were also enhanced.

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T1 jjranyi Alterations in brain extracellular dopamine and glycine levels following combined administration of the glycine transporter type-1 inhibitor Org and risperidone. Our data indicate that coadministration of an antipsychotic with a GlyT-1 inhibitor may normalize hypofunctional NMDA receptor-mediated glutamatergic neurotransmission with reduced dopaminergic side effects characteristic for antipsychotic medication. The therapeutic zsoolt of all atypical antipsychotics is explained in part by antagonism of the dopaminergic neurotransmission, mainly by blockade of D2 dopamine receptors.

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Risperidone increased extracellular concentrations of dopamine but failed to influence those of glycine or glutamate measured in microdialysis samples.

When risperidone and Org were added in combination, a decrease in extracellular dopamine concentrations was accompanied with sustained elevation of extracellular glycine levels. The most dominant hypotheses for the pathogenesis of jurany have focused primarily upon hyperfunctional dopaminergic and hypofunctional glutamatergic neurotransmission in the central nervous system.

juraniy To investigate this type of combined drug administration, rats were treated with the atypical antipsychotic risperidone together with the GlyT-1 inhibitor Org Keywords Antipsychotic agents Extracellular glycine and dopamine Glycine transporter type-1 inhibitors Microdialysis Schizophrenia.

Org injection reduced extracellular dopamine concentrations and elevated extracellular glycine levels but the concentrations of serine and glutamate were not changed.

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N2 – The most dominant hypotheses for the pathogenesis of schizophrenia have focused primarily upon hyperfunctional dopaminergic and hypofunctional glutamatergic neurotransmission in the central nervous system. Link to publication in Scopus.

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Neurochemical Research35 12 N-methyl-d-aspartate NMDA receptor hypofunction in schizophrenia can be reversed by glycine transporter type-1 GlyT-1 inhibitors, which regulate glycine concentrations at the vicinity of NMDA receptors. Access to Document Combined drug administration with D 2 dopamine receptor blockade and activation of hypofunctional NMDA receptors may be needed for a more effective treatment of positive and negative symptoms and the accompanied cognitive deficit in schizophrenia.

The therapeutic efficacy of all atypical antipsychotics is explained in part by antagonism of the dopaminergic neurotransmission, mainly by blockade of D 2 dopamine receptors.

Abstract The most dominant hypotheses for the pathogenesis of schizophrenia have focused primarily upon hyperfunctional dopaminergic and hypofunctional glutamatergic neurotransmission in the central nervous system.